Back to AI Flashcard MakerNursing /DHG 130 - Exam 1 CH 36 Fluorides Part 2
DHG 130 - Exam 1 CH 36 Fluorides Part 2
This deck covers key concepts related to fluorides, including mineral formulas, fluoride effects, and toxicity. It is designed to help students understand the role of fluoride in dental health and its systemic implications.
A group of minerals of the general formula Ca10 (PO4) X2 wherein the X might include hydroxyl (OH), carbonat (CO), fluoride (F) or oxygen (O) crystalline mineral component o hard tissue (bones and teeth)?
Apatite
Tap or swipe ↕ to flip
Swipe ←→Navigate
1/27
Key Terms
Term
Definition
A group of minerals of the general formula Ca10 (PO4) X2 wherein the X might include hydroxyl (OH), carbonat (CO), fluoride (F) or oxygen (O) crystalline mineral component o hard tissue (bones and teeth)?
Apatite
Breakdown of the tooth structure with a loss of mineral content, primarily calcium and phosphorus?
Demineralization
Decayed, missing, and filled teeth (permanent and primary dentition, respectively)?
DMFT/dmft
The form of hydroxyapatite in which fluoride ions have replaced some of the hydroxyl ions; with fluoride, the apatite is less soluble and therefore more resistant to the acids formed from carbohydrate intake?
Fluorapatite
Form of enamel hypomineralization due to excessive of fluoride during the development and mineralization of the teeth; depending on the length of exposure and the concentration of the FL2; the fluorosed area may appear as a small white spot or as severe brown staining with pitting?
Fluorosis
NaF stands for?
Neutral Sodium Fluoride
Related Flashcard Decks
Study Tips
- Press F to enter focus mode for distraction-free studying
- Review cards regularly to improve retention
- Try to recall the answer before flipping the card
- Share this deck with friends to study together
| Term | Definition |
|---|---|
A group of minerals of the general formula Ca10 (PO4) X2 wherein the X might include hydroxyl (OH), carbonat (CO), fluoride (F) or oxygen (O) crystalline mineral component o hard tissue (bones and teeth)? | Apatite |
Breakdown of the tooth structure with a loss of mineral content, primarily calcium and phosphorus? | Demineralization |
Decayed, missing, and filled teeth (permanent and primary dentition, respectively)? | DMFT/dmft |
The form of hydroxyapatite in which fluoride ions have replaced some of the hydroxyl ions; with fluoride, the apatite is less soluble and therefore more resistant to the acids formed from carbohydrate intake? | Fluorapatite |
Form of enamel hypomineralization due to excessive of fluoride during the development and mineralization of the teeth; depending on the length of exposure and the concentration of the FL2; the fluorosed area may appear as a small white spot or as severe brown staining with pitting? | Fluorosis |
NaF stands for? | Neutral Sodium Fluoride |
Occurs when foods and beverages processed in a fluoridated community are imported and consumed in a nonfluoridated community? | Halo or diffusion effect |
Measure used to designate the amount of FL2 used for optium level in FL2 water, dentifrice and other FL2 containing preparations? | ppm |
SnF2 Stands for? | Stannous Fluoride |
Restoration of mineral elements in a tooth surface; enhanced by the presence of FL2; remineralized lesions are more resistant to initiation of dental caries than is normal tooth structure? | Remineralization |
T/F Acute Fluoride poisoning is rare | True |
Symptoms usually begins within how many minutes of ingestion of acute toxic dose? | 30 mins |
T/F 99% of the fl2 in the body is located in the mineralized tissues (teeth/bones) | True |
T/F fl2 is distributed by the plasma to all tissues and organs | True |
In utero, hard tissue formation occurs during the? | 2nd trimester |
Results in the development of shallower occlusal grooves, reducing the rick of pit and fissure caries is because of the? | Pre-eruptive |
Excessive fl2 during enamel formation and tooth development results in? | Dental fluorosis, the enamel is hypomineralized |
Action of fl2 is? | Inhibits demineralization Enhance remineralization of incipient lesions Works as a desenitivation Inhibits bacterial activity by inhibiting enolase to metabolize carbs Inhibits erosion |
Most fl2 is asborbed in the? | Stomach as hydrogen fl2 |
Signs and symptoms of acute toxic dose? | Nausea vomiting diarrhea abdominal pain increased salivation thirst |
Refers to rapid intake of an excess dose over a short time? | Acute Toxicity |
Applies to long term ingestion of the fluoride in amounts that exceed the approved therapeutic levels? | Chronic toxicity |
T/F Accidental ingestion of a concentrated fluoride preparation can lead to a toxic reaction | True |
Systemic involvements of ingested fluoride? | blood; hypocalcemia CNS; hyperreflexia, convulsions, parathesis cardiac failure, respiratory paralysis |
Chronic toxicity can lead to? | Skeletal fluorosis; stiff/painful joints and becoming crippling in later stage |
5% NaF remains in teeth for number of hours releasing fl2 into what areas? | Pits and fissures | Interproximal/cervical areas…where it is needed the most |
Which fl2 reduces demineralization in white areas around orthodox brackets? | 5% NaF |