Back to AI Flashcard MakerPsychology /Psychotherapy: 87b - Circadian Rhythms and Sleep

Psychotherapy: 87b - Circadian Rhythms and Sleep

Psychology26 CardsCreated about 2 months ago

This flashcard set covers key concepts in sleep and circadian biology, including the EEG hallmark of Stage N1 sleep (theta waves indicating light sleep) and the suprachiasmatic nucleus (SCN) as the body’s primary circadian pacemaker. The SCN receives light input via the retinohypothalamic tract, aligning physiological processes with the external light-dark cycle.

What EEG pattern will you see if a patient is in Stage N1 sleep?

Theta waves

In general, the waves are slowing down

N1 = “light sleep”

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Key Terms

Term
Definition

What EEG pattern will you see if a patient is in Stage N1 sleep?

Theta waves

In general, the waves are slowing down

N1 ...

Where is the primary circaidan pacemaker located?

From where does it receive input?

Suprachiasmatic nucleus in the hypothalamus

Receives photic input from...

What EEG pattern will you see if a patient is in Stage N3 sleep?

Larger slower waves at delta frequency (5-6 Hz)

What is the FDA-approed medication for the treatment of non-24h sleep wake disorder/

Hetlioz (tasimelteon)

In general, what is the treatment for circaidan rhythm sleep disorders?

Timed light and/or melatonin exposure

Planned sleep schedules

Describe the molecular clock and associated feedback loop

The following process takes ~24h, thus providing the basis for our intrinsic circadian rhythm

  • CLOCK and BMAL...

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TermDefinition

What EEG pattern will you see if a patient is in Stage N1 sleep?

Theta waves

In general, the waves are slowing down

N1 = “light sleep”

Where is the primary circaidan pacemaker located?

From where does it receive input?

Suprachiasmatic nucleus in the hypothalamus

Receives photic input from the retinohypothalamic tract

What EEG pattern will you see if a patient is in Stage N3 sleep?

Larger slower waves at delta frequency (5-6 Hz)

What is the FDA-approed medication for the treatment of non-24h sleep wake disorder/

Hetlioz (tasimelteon)

In general, what is the treatment for circaidan rhythm sleep disorders?

Timed light and/or melatonin exposure

Planned sleep schedules

Hypnotics are not recommended; they can help with sleep, but will not re-set the circadian rhythm

Describe the molecular clock and associated feedback loop

The following process takes ~24h, thus providing the basis for our intrinsic circadian rhythm

  • CLOCK and BMAL1 are synthesized

  • They exit the nucleus and dimerize

  • CLOCK/BMAL1 dimer acts as a transcription factor that promotes the transcription of PER and CRY

  • They exit the nucelus and dimerize

  • They are phosphorylated by CK1ε/δ​

    • This is the rate limiting step

  • Phosphorylated PER/CRY dimer acts as a transcription factor that inhibits its own transcription

  • When enough Phosphorylated PER/CRY gets into the nucleus transcription of PER and CRY stops, thus allowing it to start being transcribec again
    *

The VLPO [activates/inhibits] orexinergic nuclei

This stabilizes [wakefulness/sleep]

The VLPO inhibits orexinergic nuclei

This stabilizes sleep

The [ARAS/VLPO] dominates when we are asleep, while the [ARAS/VLPO] dominates when we are awake

The VLPO dominates when we are asleep, while the ARAS ​dominates when we are awake

When does the “biological morning” start?

After the core body temperature minimum (which is usually ~2h before wake)

Which neuromodulator stabilizes the “awake” state?

Orexin (aka hypocretin)

@scientists y tho ¯_(-_-)

Inhibiting the VLPO results in [sleep/wakefulness]

Inhibiting the VLPO results in wakefulenss

  • The VLPO usually inhibits wake-promoting nuclei

Orexinergic nuclei stabilize the [ARAS/VLPO]

This results in [wakefulness/sleep]

Orexinergic nuclei stabilize the ARAS

This results in wakefulness

  • The VLPO inhibits orexinergic nuclei, resulting in sleep

When do you give melatonin to treat:

  • Delayed sleep/wake disorder:

  • Non-24h sleep/wake disorder:

  • Delayed sleep/wake disorder:

    • Give melatonin ~5h before bedtime

  • Non-24h sleep/wake disorder:

    • Give melatonin ~1h before bedtime

What EEG pattern will you see if a patient is in REM sleep?

Theta waves (just like Stage N1 sleep), may also see beta waves (like awake state)

But also:

  • Atonia

  • Rapid eye movements behind closed eyelids

Which neurotransmitters are most active in wakefulness?

ACh

Monoamines (NE, Serotonin, Dopamine)

Orexin

What EEG pattern will you see if a patient is in Stage N2 sleep?

Sleep spindles

K complexes

Theta waves will still be present

Describe the two process model that controls sleep

Process S (sleep) and Process C (circadian) work together

  • Process S

    • Homeostatic sleep need

    • The longer we are awake, the more we need to sleep

  • Proces C

    • Helps us stay awake in the afternoon when our sleep drive is high, but we’re supposed to be awake

    • Falls at night, allowing process S to knock us out

Which system modulates arousal?

Ascending reticular activating system

  • Nuclei of monoamines

  • Inhibits the VLPO

Traveling from Hawaii to Chicago will result in jet lag, with symptoms similar to which circadian rhythm sleep disorder?

How would you minimize these symtoms?

Similar to delayed sleep/wake disorder

  • Light in the biological morning when you arrive in Chicago

    • After the core body temp minimum, usually about 2h before wake time

  • Melatonin ~5h before bedtime

Which neurotransmitters are most active in NREM sleep?

VLPO

Some monoamines (Serotonin, NE, Dopamine)

Which neurotransmitters are most active in REM sleep?

ACh

MCH

VLPO

Light exposure at night and melatonin in the morning [advance/delay] the circadian clock

Light exposure at night and melatonin in the morning delay the circadian clock

Note: to treat advanced sleep/wake disorder, planned sleep schedule + light exposure in the evening. Usually don’t give melatoin in the morning

Light exposure in the biological morning and melatonin at night [advance/delay] the circadian clock

Light exposure in the biological morning and melatonin at night advance the circadian clock

ACh is active in [Wakefulness/NREM sleep/REM sleep]

ACh is active in wakefulnes and REM sleep

What EEG pattern will you see if a patient is in "relaxed wakefulness?"

Alpha waves

REM sleep occurs when monoamines levels are [low/high] and ACh levels are [low/high]

REM sleep occurs when monoamines levels are low and ACh levels are high

MCH and VLPO activity are also high